We are very fortunate to have the human malaria challenge model to take the critical next step evaluating the efficacy of GAP3KO in preventing malaria in people…
They are fortunate to be able to monetize their findings without mention of the facts already known about hemoglobin variants in hummingbirds and humans.
My comment on the Labroots report:
Hemoglobin S is the variant that links ecological variation from natural information processing and natural selection for energy-dependent codon optimality to ecological adaptation via protection from genomic entropy in human populations where malaria is endemic. More than 1200 other human hemoglobin variants link the hemoglobin variants in hummingbirds via the conserved molecular mechanisms of energy-dependent RNA-mediated cell type differentiation. Neo-Darwinian theorists have placed everything known about the variants into the context of mutation-driven evolution.
Their choice of a video representation:
The researchers failed to link energy-dependent changes in chirality to the de novo creation of the innate immune system. They also failed to link epigenetically-effected information processing and natural selection for codon optimality from autophagy to chromosomal rearrangements and all biodiversity. The chromosomal rearrangements link ecological variation from the energy-dependent physiology of pheromone-controlled reproduction to ecological adaptations in all living genera.
The facts that link all invertebrates to all vertebrates are not included in the video representation. My video representation:
Misrepresentation of facts about ecological adaptations in hummingbirds and humans fail to link nutrient energy-dependent fixation of RNA-mediated amino acid substitutions from the physiology of pheromone-controlled reproduction in all living genera to the hemoglobin variants in all vertebrates.
For example, hemoglobin S is the naturally occurring variant that links fixation of a single amino acid substitution in the organized genomes of some human populations to protection from becoming extinct.
That fact explains why these researchers are developing a vaccine to protect human populations from many different types of pathology. See: Generation of influenza A viruses as live but replication-incompetent virus vaccines